Spencer Shipman shares his biology experiment using clove and oregano on the growth of E Coli verses Amoxicillin
Video of Biology Experiment: E Coli vs Clove & Oregano
I am a holistic health practitioner and educator, nutritional counselor, herbal consultant, zyto compass analyst, therapist in biomodulator and transducer therapies and AromaTouch therapist. My goal is to teach every person to be their own healer and to make educated choices about what they put in their body. I believe plant therapies and therapeutic grade essential oils are the perfect complement to my philosophies about health.
Spencer Shipman shares his biology experiment using clove and oregano on the growth of E Coli verses Amoxicillin
The average human digestive tract is home to as many as 1,000 species of microorganisms. Most of them are harmless — or even helpful — under normal circumstances. But when something upsets the balance of these organisms in your gut, otherwise harmless bacteria can grow out of control and make you sick. One of the worst offenders is a bacterium called Clostridium difficile(C. difficile, or C. diff). As the bacteria overgrow they release toxins that attack the lining of the intestines, causing a condition called Clostridium difficilecolitis.
C. difficile infection can range from mild to life-threatening. Symptoms include watery diarrhea, with abdominal pain or tenderness, and may include loss of appitite, fever, blood or puss in the stool, weight loss.
C. difficile is labeled as an Urgent Threat by the CDC becasue it is resistant to antibiotics.
Deaths related to C. difficile increased 400% between 2000 and 2007, in part because of a stronger strain.
Most infections are connected to receiving medical care.
Hand sanitizer does not kill C. difficile, and hand washing may not be sufficient.
There is research validating that essential oils are effective against this antibiotic resistant bacteria: Cinnamon and Oregano:
The effect of cinnamon – trans-cinnamaldehyde (TC) and oregano -carvacrol (CR) on a codY mutant and wild type C. difficile was also investigated. Carvacrol and TC substantially reduced C. difficile toxin production and cytotoxicity on Vero cells. The plant compounds also significantly down-regulated toxin production genes. Carvacrol and TC did not inhibit toxin production in the codY mutant of C. difficile, suggesting a potential codY-mediated anti-toxigenic mechanism of the plant compounds. The antitoxigenic concentrations of CR and TC did not inhibit the growth of beneficial gut bacteria. Our results suggest that CR and TC could potentially be used to control C. difficile, and warrant future studies in vivo.